Yiguo zhang (35 resultados)

Condición: Very Good. Very Good condition. A copy that may have a few cosmetic defects. May also contain light spine creasing or a few markings such as an owner's name, short gifter's inscription or light stamp.

Paperback. Condición: Very Good. No Jacket. May have limited writing in cover pages. Pages are unmarked. ~ ThriftBooks: Read More, Spend Less.

Softcover. Condición: Very Good. xi, 154, [2] pp., illustrated, 4to, card wrapper. Catalogue of an exhibition held April 15 to June 6, 1998; numerous illustrations. With a presentation inscription from the author on the title page. Design and typography by Jerry Kelly. Printed on Mohawk Superfine paper at the Stinehour Press. Very good copy; small light bump to lower corner, spine mildly age-toned.…

Condición: Good. Former library copy. Pages intact with minimal writing/highlighting. The binding may be loose and creased. Dust jackets/supplements are not included. Includes library markings. Stock photo provided. Product includes identifying sticker. Better World Books: Buy Books. Do Good.

Condición: Very Good. Former library copy. Pages intact with possible writing/highlighting. Binding strong with minor wear. Dust jackets/supplements may not be included. Includes library markings. Stock photo provided. Product includes identifying sticker. Better World Books: Buy Books. Do Good.

Trade Paperback. Condición: Good. First Edition. A Good copy of the First Edition with an uncreased spine, some darkening on the spine, slight dampstaining along the top and bottom edges, and some shelfwear.

Soft cover. Condición: Very Good. No Jacket. 1st Edition. Lightly soiled copy, unmarked, in very good condition. 154 pages.

Paperback. Condición: Very Good. illustrations, xi, 155p. Softcover in original wrappers. 28 cm. Published in conjunction with the similarly titled exhibition at the gallery.

Paperback. Condición: Very Good. Exhibition catalog. Illustrated. First edition (paperback). Very good in oversize printed wraps.

Soft cover. Condición: Near Fine. 1st Edition. SOFT COVER IN NEAR FINE CONDITION.

Paperback. Condición: New. New - unused and unread. First Edition. Faint shelf wear.

Paperback or Softback. Condición: New. Nrf1, serving as a Highly-Conserved Determinon for Robust Redox Homeostasis. Book.

Paperback. Condición: Very Good. First Edition. New York: Wallach Art Gallery, Columbia University, 1998. First edition, 1998. Softcover, 156 pages. Some minor shelf soil to the white covers, firm binding, clean pages, no names or other markings. First Edition. Soft Cover. Very Good. 4to - over 9¾" - 12" tall. …

Condición: New.

Hardcover. Condición: New. Estado de la sobrecubierta: New. 1st Edition. Olive-brown cloth, lettered in gold foil. New/as issued, dust jacket now in mylar. 1st ptg. ix,174 pp., illus. Size: Folio - over 12" - 15" tall. Book.

Soft cover. Condición: Very Good. Trade paperback in very good + condition.

Softcover. Condición: VG. Off-white illus. wraps; 154 pp.; 11 color plates; 65+ bw figures. Accompanied the exhibition of the same name held at the Miriam & Ira D. Wallach Art Gallery at Columbia University; Includes calligraphy by Bai Di, Liu Bingsen, Luo Qi, Shao Yan, Wang Yong, Zhou Huijan, and others.…

Paperback. Condición: Brand New. 106 pages. 6.00x0.25x9.00 inches. In Stock.

Softcover. Condición: Very good. No jacket. Mild shelfwear.

paperback. Condición: New. In shrink wrap. Looks like an interesting title.

Taschenbuch. Condición: Neu. Molecular and Cellular Control of the Nrf1 Transcription Factor | An Integral Membrane Glycoprotein | Yiguo Zhang | Taschenbuch | Englisch | VDM Verlag Dr. Müller | EAN 9783639148411 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | Anbieter: preigu. …

Condición: New.

Cloth, slipcase. Condición: Good, unless otherwise noted. 31x24cm, 287 pp. Numerous full page colour plates throughout. // Catalogue of an American collection of Ming, Qing and 20th century Chinese painting and calligraphy which has been formed over the last 20 years. All illustrated in colour and well-described. Dual text in English and Chinese.…

Condición: New. Print on Demand.

Condición: New. Print on Demand.

Condición: New. PRINT ON DEMAND.

Taschenbuch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware 108 pp. Englisch.

Taschenbuch. Condición: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Differential and even opposing functions of two major antioxidant transcription factors Nrf1 and Nrf2 (encoded by Nfe2l1 and Nfe2l2, respectively) are determined by distinctions in their tempospatial positioning, topological repartitioning, proteolytic processing, and biochemical modification, as well as in their shared evolutionary origin. As a matter of fact, the allelopathic potentials of Nrf1 and Nrf2 (both resembling two entangled 'Yin-Yang' quanta that comply with a dialectic law of the unity of opposites) are fulfilled to coordinately control redox physiological homeostasis so as to be maintained within the presetting thresholds. By putative exponential curves of redox stress and intrinsic anti-redox capability, there is inferable to exist a set point at approaching zero with the 'Golden Mean' for the healthy survival (i.e., dubbed the 'zero theory'). A bulk of the hitherto accumulating evidence demonstrates that the set point of redox homeostasis is dictated selectively by multi-hierarchical threshold settings, in which the living fossil-like Nrf1 acts as a robust indispensable determinon, whereas Nrf2 serves as a versatile chameleon-like master regulon, in governing the redox homeodynamic ranges. This is attributable to the facts that Nrf2 has exerted certain 'double-edged sword' effects on life process, whereas Nrf1 executes its essential physiobiological functions, along with unique pathophysiological phenotypes, by integrating its 'three-in-one' roles elicited as a specific triplet of direct sensor, transducer and effector within multi-hierarchical stress responsive signaling to redox metabolism and target gene reprogramming. Here, we also critically reviewed redox regulation of physio-pathological functions from the eco-evo-devo perspectives, through those coding rules (redox code, stress-coping code, and topogenetic code). The evolving concepts on stress and redox stress were also further revisited by scientific principles of physics and chemistry, apart from two novel concepts of 'oncoprotists' and 'reverse central dogma' being introduced in this interdisciplinary and synthetic review.…

Paperback. Condición: new. Paperback. Differential and even opposing functions of two major antioxidant transcription factors Nrf1 and Nrf2 (encoded by Nfe2l1 and Nfe2l2, respectively) are determined by distinctions in their tempospatial positioning, topological repartitioning, proteolytic processing, and biochemical modification, as well as in their shared evolutionary origin. As a matter of fact, the allelopathic potentials of Nrf1 and Nrf2 (both resembling two entangled 'Yin-Yang' quanta that comply with a dialectic law of the unity of opposites) are fulfilled to coordinately control redox physiological homeostasis so as to be maintained within the presetting thresholds. By putative exponential curves of redox stress and intrinsic anti-redox capability, there is inferable to exist a set point at approaching zero with the 'Golden Mean' for the healthy survival (i.e., dubbed the 'zero theory'). A bulk of the hitherto accumulating evidence demonstrates that the set point of redox homeostasis is dictated selectively by multi-hierarchical threshold settings, in which the living fossil-like Nrf1 acts as a robust indispensable determinon, whereas Nrf2 serves as a versatile chameleon-like master regulon, in governing the redox homeodynamic ranges. This is attributable to the facts that Nrf2 has exerted certain 'double-edged sword' effects on life process, whereas Nrf1 executes its essential physiobiological functions, along with unique pathophysiological phenotypes, by integrating its 'three-in-one' roles elicited as a specific triplet of direct sensor, transducer and effector within multi-hierarchical stress responsive signaling to redox metabolism and target gene reprogramming. Here, we also critically reviewed redox regulation of physio-pathological functions from the eco-evo-devo perspectives, through those coding rules (redox code, stress-coping code, and topogenetic code). The evolving concepts on stress and redox stress were also further revisited by scientific principles of physics and chemistry, apart from two novel concepts of 'oncoprotists' and 'reverse central dogma' being introduced in this interdisciplinary and synthetic review. This item is printed on demand. Shipping may be from our UK warehouse or from our Australian or US warehouses, depending on stock availability.…

Taschenbuch. Condición: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Differential and even opposing functions of two major antioxidant transcription factors Nrf1 and Nrf2 (encoded by Nfe2l1 and Nfe2l2, respectively) are determined by distinctions in their tempospatial positioning, topological repartitioning, proteolytic processing, and biochemical modification, as well as in their shared evolutionary origin. As a matter of fact, the allelopathic potentials of Nrf1 and Nrf2 (both resembling two entangled 'Yin-Yang' quanta that comply with a dialectic law of the unity of opposites) are fulfilled to coordinately control redox physiological homeostasis so as to be maintained within the presetting thresholds. By putative exponential curves of redox stress and intrinsic anti-redox capability, there is inferable to exist a set point at approaching zero with the 'Golden Mean' for the healthy survival (i.e., dubbed the 'zero theory'). A bulk of the hitherto accumulating evidence demonstrates that the set point of redox homeostasis is dictated selectively by multi-hierarchical threshold settings, in which the living fossil-like Nrf1 acts as a robust indispensable determinon, whereas Nrf2 serves as a versatile chameleon-like master regulon, in governing the redox homeodynamic ranges. This is attributable to the facts that Nrf2 has exerted certain 'double-edged sword' effects on life process, whereas Nrf1 executes its essential physiobiological functions, along with unique pathophysiological phenotypes, by integrating its 'three-in-one' roles elicited as a specific triplet of direct sensor, transducer and effector within multi-hierarchical stress responsive signaling to redox metabolism and target gene reprogramming. Here, we also critically reviewed redox regulation of physio-pathological functions from the eco-evo-devo perspectives, through those coding rules (redox code, stress-coping code, and topogenetic code). The evolving concepts on stress and redox stress were also further revisited by scientific principles of physics and chemistry, apart from two novel concepts of 'oncoprotists' and 'reverse central dogma' being introduced in this interdisciplinary and synthetic review.Libri GmbH, Europaallee 1, 36244 Bad Hersfeld 108 pp. Englisch.…