9783319174365 - cardiac fibrosis and heart failure: cause or effect?: 13 (advances in biochemistry in health and disease) (12 resultados)
Idioma: Inglés
Editorial: Springer, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: Ria Christie Collections, Uxbridge, Reino UnidoRia Christie Collections
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Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: moluna, Greven, Alemaniamoluna
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Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: Books Puddle, New York, NY, Estados Unidos de AmericaBooks Puddle
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Condición: New. pp. 746.
Idioma: Inglés
Editorial: Springer, Springer, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: AHA-BUCH GmbH, Einbeck, AlemaniaAHA-BUCH GmbH
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EUR 160,49
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Buch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - The unique biology of cardiac fibroblasts and related cells, such as cardiac myofibroblasts and valvular interstitial cells, distinguish them from other fibroblastic cells, a concept that is only beginning to be widely appreciated. Further, the natural si…gnals that stimulate and inhibit cardiac fibrosis within these cells are not well understood. This volume compiles articles that address the molecular mechanisms that control the synthesis and secretion of the cardiac ECM. The book showcases chapters that highlight discussion of role of Transforming Growth Factor beta (TGFbeta), an important fibrogenic cytokine and its downstream effectors SMAD in many cardiac diseases. Further, the contributions highlight information to discuss endogenous inhibitors of cardiac fibrosis, as well as advances in tissue engineering specific to matrix in the heart. Finally, discussions of unifying mechanisms of matrix remodeling in valves and myocardium are presented.The mechanisms involved in the stimulation of cardiac fibrosis are not fully understood. In most cases the marginal attenuation of cardiac fibrosis as a result of a given therapy is a beneficial side-effect linked to other primary effects on other cells, especially cardiomyocytes. Very few drugs or agents are known to affect the function and dysfunction of cardiac fibroblasts and myofibroblasts alone. The book helps to translate the information gathered within to allow us to alter the course of fibrogenic events that are typical of cardiac fibrosis, and thereby reduce their burden on the patient and on society itself.
Idioma: Inglés
Editorial: Springer Verlag, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: Revaluation Books, Exeter, Reino UnidoRevaluation Books
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EUR 239,23
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Hardcover. Condición: Brand New. 2015 edition. 454 pages. 9.25x6.10x1.10 inches. In Stock.
Idioma: Inglés
Editorial: Springer, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: Mispah books, Redhill, SURRE, Reino UnidoMispah books
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Hardcover. Condición: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.
Idioma: Inglés
Editorial: Springer, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: Brook Bookstore On Demand, Napoli, NA, ItaliaBrook Bookstore On Demand
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EUR 126,26
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Condición: new. Questo è un articolo print on demand.
Idioma: Inglés
Editorial: Springer, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: Basi6 International, Irving, TX, Estados Unidos de AmericaBasi6 International
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EUR 137,58
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Condición: Brand New. New. US edition. Print on demand title. Delivery takes 20-25 days. Excellent Customer Service.
Idioma: Inglés
Editorial: Springer International Publishing Jul 2015, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, AlemaniaBuchWeltWeit Ludwig Meier e.K.
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EUR 160,49
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Buch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -The unique biology of cardiac fibroblasts and related cells, such as cardiac myofibroblasts and valvular interstitial cells, distinguish them from other fibroblastic cells, a concept that is only beginning to be widely appreciated. Further…, the natural signals that stimulate and inhibit cardiac fibrosis within these cells are not well understood. This volume compiles articles that address the molecular mechanisms that control the synthesis and secretion of the cardiac ECM. The book showcases chapters that highlight discussion of role of Transforming Growth Factor beta (TGFbeta), an important fibrogenic cytokine and its downstream effectors SMAD in many cardiac diseases. Further, the contributions highlight information to discuss endogenous inhibitors of cardiac fibrosis, as well as advances in tissue engineering specific to matrix in the heart. Finally, discussions of unifying mechanisms of matrix remodeling in valves and myocardium are presented.The mechanisms involved in the stimulation of cardiac fibrosis are not fully understood. In most cases the marginal attenuation of cardiac fibrosis as a result of a given therapy is a beneficial side-effect linked to other primary effects on other cells, especially cardiomyocytes. Very few drugs or agents are known to affect the function and dysfunction of cardiac fibroblasts and myofibroblasts alone. The book helps to translate the information gathered within to allow us to alter the course of fibrogenic events that are typical of cardiac fibrosis, and thereby reduce their burden on the patient and on society itself. 452 pp. Englisch.
Idioma: Inglés
Editorial: Springer, Springer Jul 2015, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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- Impresión bajo demanda
Librería: buchversandmimpf2000, Emtmannsberg, BAYE, Alemaniabuchversandmimpf2000
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EUR 160,49
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Buch. Condición: Neu. This item is printed on demand - Print on Demand Titel. Neuware -The unique biology of cardiac fibroblasts and related cells, such as cardiac myofibroblasts and valvular interstitial cells, distinguish them from other fibroblastic cells, a concept that is only beginning to be widely appreciated. Further, th…e natural signals that stimulate and inhibit cardiac fibrosis within these cells are not well understood. This volume compiles articles that address the molecular mechanisms that control the synthesis and secretion of the cardiac ECM. The book showcases chapters that highlight discussion of role of Transforming Growth Factor ¿ (TGF¿), an important fibrogenic cytokine and its downstream effectors SMAD in many cardiac diseases. Further, the contributions highlight information to discuss endogenous inhibitors of cardiac fibrosis, as well as advances in tissue engineering specific to matrix in the heart. Finally, discussions of unifying mechanisms of matrix remodeling in valves and myocardium are presented.The mechanisms involved in the stimulation of cardiac fibrosis are not fully understood. In most cases the marginal attenuation of cardiac fibrosis as a result of a given therapy is a beneficial side-effect linked to other primary effects on other cells, especially cardiomyocytes. Very few drugs or agents are known to affect the function and dysfunction of cardiac fibroblasts and myofibroblasts alone. The book helps to translate the information gathered within to allow us to alter the course of fibrogenic events that are typical of cardiac fibrosis, and thereby reduce their burden on the patient and on society itself.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 452 pp. Englisch.
Idioma: Inglés
Editorial: Springer, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
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Librería: Majestic Books, Hounslow, Reino UnidoMajestic Books
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EUR 223,62
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Condición: New. Print on Demand pp. 746.
Idioma: Inglés
Editorial: Springer, 2015
Serie: Libro 10 de 25 - Advances in Biochemistry in Health and Disease
- Tapa dura
- Impresión bajo demanda
Librería: Biblios, frankfurt am main, HESSE, AlemaniaBiblios
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EUR 226,79
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Condición: New. PRINT ON DEMAND pp. 746.
