Isbn: 9780792362784 - myasthenia gravis: disease mechanism and immunointervention (14 resultados)

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  • Idioma: Inglés

    Editorial: Springer Netherlands, 2000

    0792362780 / 9780792362784

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    Librería: Better World Books, Mishawaka, IN, Estados Unidos de AmericaBetter World Books

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    Condición: Usado - Aceptable

    EUR 96,39

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    Condición: Good. Former library copy. Pages intact with minimal writing/highlighting. The binding may be loose and creased. Dust jackets/supplements are not included. Includes library markings. Stock photo provided. Product includes identifying sticker. Better World Books: Buy Books. Do Good.

  • Idioma: Inglés

    Editorial: Springer, 2000

    0792362780 / 9780792362784

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    Librería: Ria Christie Collections, Uxbridge, Reino UnidoRia Christie Collections

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    EUR 128,96

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    Condición: New. In English.

  • Idioma: Inglés

    Editorial: Kluwer Academic Publishers, 2000

    0792362780 / 9780792362784

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    Librería: Kennys Bookshop and Art Galleries Ltd., Galway, GY, IrlandaKennys Bookshop and Art Galleries Ltd.

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    EUR 130,33

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    Cantidad disponible: 15 disponibles

    Condición: New. Autoimmune myasthenia gravis (MG) is a classical autoimmune disease, for which the target antigen, nicotinic acetylcholine receptor, has been cloned, sequenced and biochemically characterized. This book is the outcome of the MG workshop proceedings in Mysore, India, 1998. It contains reviews of specific topics and also of various findings. Editor(s): Christadoss, Premkumar. Num Pages: 208 pages, biography. BIC Classification: MJC; MJN. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 235 x 155 x 14. Weight in Grams: 550. . 2000. Hardback. . . . .…

  • Idioma: Inglés

    Editorial: Springer, 2000

    0792362780 / 9780792362784

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    Librería: AHA-BUCH GmbH, Einbeck, AlemaniaAHA-BUCH GmbH

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    Condición: Nuevo

    EUR 114,00

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    Cantidad disponible: 1 disponible

    Buch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - Autoimmune myasthenia gravis (MG) is a classical autoimmune disease, for which the target antigen, nicotinic acetylcholine receptor, has been cloned, sequenced and biochemically characterized. Antibodies to acetylcholine receptors destroy acetylcholine receptor at the neuromuscular junction, thus leading to defective neuromuscular transmission, muscle fatigue, and weakness. In the last few years, rapid advances have been made in unraveling the cellular and molecular mechanisms involved in the pathogenesis of MG, both in the animal model, experimental autoimmune MG (EAMG), and in human MG. Significant advances are being made in characterizing the cells and molecules involved in the autoimmune response to the acetylcholine receptor (AChR). These advances are leading to the development of specific methods of immunointervention in EAMG. Further understanding of the intricate involvement of the major histocompatibility complex (MHC) and non-MHC genes, T cell receptors (TCR), costimulator molecules, and specific cytokines in the afferent and efferent autoimmune response of AChR should pave the way to future antigen/clone-specific therapy of MG. This book is the outcome of the MG workshop proceedings in Mysore, India, 1998. The majority of the chapters in this book are contributed by world-renowned authors and their students. The book not only contains a timely review of specific topics, but also up-to-date findings. Immunologists and neurologists will find, in this volume, the latest in MG/EAMG cutting-edge research. Clinicians will be interested in the applications of the various immunointervention strategies into clinical trials in MG patients. Finally, students will not only be interested in reading the latest in EAMG/MG research, but will also find information to help them develop a future strategy to unravel the precise mechanism of disease. To summarize, in this book, the readers should find up-to-date information related to immunological mechanisms involved in MG pathogenesis and various modalities for possible approaches to immunointervention to treat MG.…

  • Idioma: Inglés

    Editorial: Springer, 2000

    0792362780 / 9780792362784

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    Librería: Books Puddle, Woodside, NY, Estados Unidos de AmericaBooks Puddle

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    Condición: Nuevo

    EUR 159,40

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    Cantidad disponible: 4 disponibles

    Condición: New. pp. 228.

  • Idioma: Inglés

    Editorial: Kluwer Academic Pub, 2000

    0792362780 / 9780792362784

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    Librería: Revaluation Books, Exeter, Reino UnidoRevaluation Books

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    Condición: Nuevo

    EUR 158,09

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    Cantidad disponible: 2 disponibles

    Hardcover. Condición: Brand New. 208 pages. 9.25x6.25x0.75 inches. In Stock.

  • Idioma: Inglés

    Editorial: Kluwer Academic Publishers, 2000

    0792362780 / 9780792362784

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    Librería: Kennys Bookstore, Olney, MD, Estados Unidos de AmericaKennys Bookstore

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    Condición: Nuevo

    EUR 167,50

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    Cantidad disponible: 15 disponibles

    Condición: New. Autoimmune myasthenia gravis (MG) is a classical autoimmune disease, for which the target antigen, nicotinic acetylcholine receptor, has been cloned, sequenced and biochemically characterized. This book is the outcome of the MG workshop proceedings in Mysore, India, 1998. It contains reviews of specific topics and also of various findings. Editor(s): Christadoss, Premkumar. Num Pages: 208 pages, biography. BIC Classification: MJC; MJN. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 235 x 155 x 14. Weight in Grams: 550. . 2000. Hardback. . . . . Books ship from the US and Ireland.…

  • Idioma: Inglés

    Editorial: Springer, 2000

    0792362780 / 9780792362784

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    Librería: Mispah books, Redhill, SURRE, Reino UnidoMispah books

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    Condición: Usado - Como Nuevo

    EUR 194,57

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    Hardcover. Condición: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.

  • Idioma: Inglés

    Editorial: Springer Netherlands Aug 2000, 2000

    0792362780 / 9780792362784

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    Librería: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, AlemaniaBuchWeltWeit Ludwig Meier e.K.

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    Condición: Nuevo

    EUR 106,99

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    Buch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Autoimmune myasthenia gravis (MG) is a classical autoimmune disease, for which the target antigen, nicotinic acetylcholine receptor, has been cloned, sequenced and biochemically characterized. Antibodies to acetylcholine receptors destroy acetylcholine receptor at the neuromuscular junction, thus leading to defective neuromuscular transmission, muscle fatigue, and weakness. In the last few years, rapid advances have been made in unraveling the cellular and molecular mechanisms involved in the pathogenesis of MG, both in the animal model, experimental autoimmune MG (EAMG), and in human MG. Significant advances are being made in characterizing the cells and molecules involved in the autoimmune response to the acetylcholine receptor (AChR). These advances are leading to the development of specific methods of immunointervention in EAMG. Further understanding of the intricate involvement of the major histocompatibility complex (MHC) and non-MHC genes, T cell receptors (TCR), costimulator molecules, and specific cytokines in the afferent and efferent autoimmune response of AChR should pave the way to future antigen/clone-specific therapy of MG. This book is the outcome of the MG workshop proceedings in Mysore, India, 1998. The majority of the chapters in this book are contributed by world-renowned authors and their students. The book not only contains a timely review of specific topics, but also up-to-date findings. Immunologists and neurologists will find, in this volume, the latest in MG/EAMG cutting-edge research. Clinicians will be interested in the applications of the various immunointervention strategies into clinical trials in MG patients. Finally, students will not only be interested in reading the latest in EAMG/MG research, but will also find information to help them develop a future strategy to unravel the precise mechanism of disease. To summarize, in this book, the readers should find up-to-date information related to immunological mechanisms involved in MG pathogenesis and various modalities for possible approaches to immunointervention to treat MG. 228 pp. Englisch.…

  • Idioma: Inglés

    Editorial: Springer Netherlands, 2000

    0792362780 / 9780792362784

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    Librería: moluna, Greven, Alemaniamoluna

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    Condición: Nuevo

    EUR 92,27

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    Gebunden. Condición: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autoimmune myasthenia gravis (MG) is a classical autoimmune disease, for which the target antigen, nicotinic acetylcholine receptor, has been cloned, sequenced and biochemically characterized. Antibodies to acetylcholine receptors destroy acetylcholine r.…

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    Idioma: Inglés

    Editorial: Springer Netherland, 2000

    0792362780 / 9780792362784

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    Librería: preigu, Osnabrück, Alemaniapreigu

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    Buch. Condición: Neu. Myasthenia Gravis | Disease Mechanism and Immunointervention | Premkumar Christadoss | Buch | xiii | Englisch | 2000 | Springer Netherland | EAN 9780792362784 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu Print on Demand. …

  • Idioma: Inglés

    Editorial: Springer, 2000

    0792362780 / 9780792362784

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    Librería: Majestic Books, Hounslow, Reino UnidoMajestic Books

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    Condición: Nuevo

    EUR 165,80

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    Condición: New. Print on Demand pp. 228.

  • Idioma: Inglés

    Editorial: Springer Netherlands, Springer Netherlands Aug 2000, 2000

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    Librería: buchversandmimpf2000, Emtmannsberg, BAYE, Alemaniabuchversandmimpf2000

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    Condición: Nuevo

    EUR 106,99

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    Buch. Condición: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Autoimmune myasthenia gravis (MG) is a classical autoimmune disease, for which the target antigen, nicotinic acetylcholine receptor, has been cloned, sequenced and biochemically characterized. Antibodies to acetylcholine receptors destroy acetylcholine receptor at the neuromuscular junction, thus leading to defective neuromuscular transmission, muscle fatigue, and weakness.In the last few years, rapid advances have been made in unraveling the cellular and molecular mechanisms involved in the pathogenesis of MG, both in the animal model, experimental autoimmune MG (EAMG), and in human MG. Significant advances are being made in characterizing the cells and molecules involved in the autoimmune response to the acetylcholine receptor (AChR). These advances are leading to the development of specific methods of immunointervention in EAMG. Further understanding of the intricate involvement of the major histocompatibility complex (MHC) and non-MHC genes, T cell receptors (TCR), costimulator molecules, and specific cytokines in the afferent and efferent autoimmune response of AChR should pave the way to future antigen/clone-specific therapy of MG.This book is the outcome of the MG workshop proceedings in Mysore, India, 1998. The majority of the chapters in this book are contributed by world-renowned authors and their students. The book not only contains a timely review of specific topics, but also up-to-date findings. Immunologists and neurologists will find, in this volume, the latest in MG/EAMG cutting-edge research. Clinicians will be interested in the applications of the various immunointervention strategies into clinical trials in MG patients. Finally, students will not only be interested in reading the latest in EAMG/MG research, but will also find information to help them develop a future strategy to unravel the precise mechanism of disease.To summarize, in this book, the readers should find up-to-date information related to immunological mechanisms involved in MG pathogenesis and various modalities for possible approaches to immunointervention to treat MG.Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 228 pp. Englisch.…

  • Idioma: Inglés

    Editorial: Springer, 2000

    0792362780 / 9780792362784

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    Librería: Biblios, frankfurt am main, HESSE, AlemaniaBiblios

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    EUR 164,31

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    Condición: New. PRINT ON DEMAND pp. 228.