K h stenzel (44 resultados)

Hardcover. Condición: Very Good. No Jacket as Issued. Original paper copy rebound in red library buckram with title in white on spine, pp 1 through 37 and 39 through 54, original paper wraps not bound in, frontis, intro, graphs, tables, fold-out map, photos, maps, profile of cut section, appendix. Scotch tap repair to gutter at pp 26/27 has turned brown and discolored page edges. Usual library marks, stickers, stamps, card pocket, etc. Rare. Newshelf 11. …

Hard Cover. Condición: Very Good+. No Jacket. The University of Texas Press, 1957, 8vo, 237 pages, illustrations, fold-outs. Hardcover edition bound in a black cloth, no dust jacket. Richard I. Johnson stamped on front endpage, book very good+ condition. Mr. Johnson was a long time Research Associate at Harvard's Museum of Comparative Zoology, having started as a volunteer while still in high school. He published his first scientific article in 1941, at the age of 16, beginning his life as a gentleman scholar, an amateur with a worldwide scholarly reputation who produced more than 50 papers about malacology. Over six decades Johnson assembled perhaps the largest private collection of books and journals on molluscs, including titles seldom found even in research libraries, each volume identified with a discrete stamp of "Richard I. Johnson" to a preliminary leaf. It was one of the greatest collections of shell books ever assembled. Size: 8vo - over 7¾" - 9¾" Tall.…

Soft cover. Condición: Very Good. SCARCE. 237pp. NO CHIPPING & NO SPLITTING to covers. A few small light marks & lightening to spine. Ink title written on spine. Internally CLEAN with NO INSCRIPTIONS. TIGHT textblock. NO LOOSE & NO MISSING leaves.

Condición: New.

Condición: New. In.

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Condición: As New. Unread book in perfect condition.

Condición: As New. Unread book in perfect condition.

Condición: New. pp. 324.

Paperback. Condición: Brand New. 324 pages. 9.30x6.00x0.90 inches. In Stock.

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Taschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - Blood pressure control is central to all bodily functions. There are many points in the multifaceted cybernetic system wherein hypertension may be produced. Hypertension is a 'young' disorder whose existence has been known for less than a century. It is not only extremely prevalent among every popula tion, but also deleterious to the health of mankind. The more we understand about hypertension's harmful effects, the more urgent is the need for its effective control. The kidney is the central organ that controls vascular tone and body fluid volume; these two factors are dominant in determining arterial blood pres sure. Hence, it is not surprising to find in hypertensive disorders that there are abnormalities in the kidneys, functional or anatomical, subtle or overt, that cause or are the consequence of hypertension. The first suggestion that the kidney could cause hypertension was made in 1836, before arterial pressure could even be measured, by Richard Bright. He observed that cardiac hypertrophy was often present in patients who died of renal disease. It was, however, Goldblatt and his colleagues in 1934 who opened the modern era of experimental and clinical research in renal hypertension. Since then, although far from complete, enthusiastic and intensive research efforts have greatly improved our understanding of the nature of renal hypertension.…

Paperback. Condición: Brand New. 1981 edition. 491 pages. 9.45x6.30x1.18 inches. In Stock.

Condición: New. Editor(s): Cheigh, Jhoong S.; Stenzel, Kurt H.; Rubin, Albert L. Series: Developments in Nephrology. Num Pages: 312 pages, biography. BIC Classification: MJR. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly. Dimension: 235 x 155 x 19. Weight in Grams: 636. . 1986. Hardback. . . . . …

Taschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - 1. Evaluation of patients with renal disease.- 1. Introduction.- 2. Identifying Renal Disease.- 2.1. History.- 2.2. Family history.- 2.3. Physical examination.- 2.4. Urinalysis.- 2.5. The chemical data base.- 2.6. Radiologic techniques.- 2.7. Percutaneous renal biopsy.- 2.8. Risks and cost of procedures.- 3. Syndromes of Renal Disease.- 3.1. Acute renal failure.- 3.2. Chronic renal failure.- 3.3. Acute nephritic syndrome.- 3.4. Nephrotic syndrome.- 3.5. Urinary tract infection.- 3.6. Obstructive nephropathy.- 3.7. Renal tubular dysfunction.- 3.8. Nephrolithiasis.- 3.9. Accelerated hypertension.- 4. Conclusion.- 2. Disorders of water, sodium and potassium metabolism.- 1. Maintenance of Osmotic Homeostasis.- 1.1. General considerations.- 1.2. Free-water clearance.- 1.3. Renal concentrating and diluting mechanisms.- 1.4. Antidiuretic hormone.- 1.5. Summary of requirements for maximum water diuresis $$left( {{C_{{H_2}O}}}ight)$$.- 2. Clinical States Associated with Hypo-osmolality.- 2.1. True dilutional hyponatremia.- 2.2. Renal failure.- 2.3. Decreased delivery of filtrate to diluting segment of the nephron.- 2.4. Syndrome of inappropriate ADH (SIADH).- 2.5 Drugs associated with impaired free-water clearance.- 2.6. Endocrine deficiencies.- 2.7. Reset osmostat.- 2.8. Summary of the diagnostic approach to hyponatremia.- 3. Hypernatremia and Disorders of Urine Concentration.- 3.1. Low circulating levels of ADH.- 3.2. Renal tubular hyporesponsiveness to ADH (nephrogenic diabetes insipidus).- 3.3. Diagnostic approach to the patient with a disorder of urinary concentration.- 4. Disorders of Sodium Metabolism.- 4.1. Determinants of renal sodium excretion.- 4.2. Sodium retaining states (edema).- 4.3. Diuretic therapy.- 5. Disorders of Potassium Metabolism.- 5.1. Internal potassium balance.- 5.2. External potassium balance.- 5.3. Hyperkalemia.- 5.4. Hypokalemia.- 3. Acid-base disturbances.- 1. Introduction.- 1.1. Physiologic buffers.- 1.2. Respiratory control of pH.- 1.3. Renal control of H + excretion.- 1.4. Clinical definitions.- 1.5. Diagnostic approach.- 2. Metabolic Acidosis.- 2.1. Anion gap.- 2.2. Metabolic acidosis with an increased anion gap.- 2.3. Metabolic acidosis associated with a normal anion gap (hyperchloremic).- 2.4. Treatment of metabolic acidosis.- 3. Metabolic Alkalosis.- 3.1. Metabolic alkalosis associated with low urinary Cl -.- 3.2. Metabolic alkalosis associated with normal or increased urinary Cl -.- 3.3. Treatment of metabolic alkalosis.- 4. Respiratory Alkalosis.- 5. Respiratory Acidosis.- 6. Mixed Acid-Base Disorders.- 4. Glomerulonephropathies.- 1. Introduction.- 2. Asymptomatic Urinary Abnormalities.- 2.1. Hematuria.- 2.2. Asymptomatic proteinuria.- 2.3. Asymptomatic proteinuria and hematuria.- 3. Nephrotic Syndrome.- 3.1. Definition.- 3.2. Pathogenesis of proteinuria.- 3.3. Patient presentation and evaluation.- 3.4. Clinicopathological correlation.- 4. Acute Nephritic Syndrome.- 4.1. Definition.- 4.2. Patient presentation and evaluation.- 4.3. Clinicopathological correlation.- 4.4. Management of acute nephritic syndrome.- 4.5. Prognosis.- 5. Rapidly Progressive Glomerulonephritis (RPGN).- 5.1. Definition.- 5.2. Patient presentation and evaluation.- 5.3. Clinicopathological correlation.- 6. Chronic Nephritic Syndrome.- 6.1. Definition.- 6.2. Patient presentation and evaluation.- 7. Glomerulonephritis and Systemic Disease.- 7.1. Metabolic and inherited diseases.- 7.2. Hereditary nephritis.- 7.3. Infectious disease.- 7.4. Toxic nephropathy.- 7.5. Collagen vascular disease.- 7.6. Dysproteinemias.- 7.7. Pregnancy.- 5. Urinary tract infection and pyelonephritis.- 1. Prevalence.- 1.1. Overall incidence.- 1.2. Prevalence of urinary tract infection in children.- 1.3. Prevalence of urinary tract infection in adults.- 2. Clinical Significance of Bacteriuria.- 2.1. Pregnancy and urinary tract infection.- 2.2. Hypertension and urinary tract infection.- 2.3. Diabetes and urinary tract infection.- 2.4. Bacteriuria in non-pregnant women.- 2.5. Bacteriuria in children.- 2.6. Bacteriuria in men.- 2.7. Nosocomial urinary tract infections.- 3. Pathogenesis.- 3.1. Local barriers of invasion.- 3.2. Cervico-vaginal antibodies.- 3.3. Uroepithelial antibodies.- 3.4. Systemic antibody response.- 3.5. Other mechanisms.- 3.6. Vesicoureteral valve.- 4. Etiology.- 4.1. Bacterial infection.- 4.2. Fungal infections.- 4.3. Viral infection.- 5. Clinical Manifestations.- 6. Diagnosis.- 6.1. Urine collection.- 6.2. Microscopic examination.- 6.3. Localization.- 7. Management.- 7.1. Asymptomatic bacteriuria.- 7.2. Acute pyelonephritis.- 7.3. Recurrent infections.- 7.4. Antibiotics.- 7.5. Prophylaxis.- 6. Tubulo-interstitial nephritis.- 1. Hereditary Kidney Diseases.- 1.1. Medullary cystic disease.- 1.2. Medullary sponge kidney.- 1.3. Hereditary familial nephritis (Alport¿s syndrome).- 2. Metabolic Kidney Diseases.- 2.1. Hypercalcemia.- 2.2. Hypokalemia.- 2.3. Oxalate nephropathy.- 2.4. Gouty nephropathy.- 3. Hematologic Diseases.- 3.1. Hemolytic-uremic syndrome.- 3.2. Sickle cell anemia.- 4. Vascular Diseases.- 5. Neoplastic Diseases.- 6. Infections.- 7. Immunological Diseases.- 7.1. Antitubular basement membrane antibodies.- 7.2. Immune complex interstitial nephritis.- 7.3. Reflux nephropathy.- 7.4. Drug-related interstitial nephritis.- 7.5. Renal allografts.- 7.6. Sj¿gren¿s syndrome.- 8. Analgesic Nephropathy.- 8.1. Aspirin.- 8.2. Phenacetin.- 8.3. Pathology.- 8.4. Pathogenesis.- 8.5. Clinical manifestations.- 9. Heavy Metals.- 10. Balkan Nephropathy.- 11. Radiation Nephritis.- 7. Cystic diseases of the kidney.- 1. Introduction.- 2. Cystic Dysplasia.- 2.1. Renal aplasia.- 2.2. Congenital multicystic kidney.- 2.3. Pathology.- 2.4. Etiology and pathogenesis.- 2.5. Clinical presentation.- 2.6. Diagnosis.- 2.7. Prognosis and therapy.- 3. Polycystic Disease.- 3.1. Infantile polycystic disease.- 3.2. Adult polycystic disease.- 4. Medullary Cysts of the Kidney. …

Condición: New. Editor(s): Cheigh, Jhoong S.; Stenzel, Kurt H.; Rubin, Albert L. Series: Developments in Nephrology. Num Pages: 312 pages, biography. BIC Classification: MJR. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly. Dimension: 235 x 155 x 19. Weight in Grams: 636. . 1986. Hardback. . . . . Books ship from the US and Ireland. …

Buch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - Blood pressure control is central to all bodily functions. There are many points in the multifaceted cybernetic system wherein hypertension may be produced. Hypertension is a 'young' disorder whose existence has been known for less than a century. It is not only extremely prevalent among every popula tion, but also deleterious to the health of mankind. The more we understand about hypertension's harmful effects, the more urgent is the need for its effective control. The kidney is the central organ that controls vascular tone and body fluid volume; these two factors are dominant in determining arterial blood pres sure. Hence, it is not surprising to find in hypertensive disorders that there are abnormalities in the kidneys, functional or anatomical, subtle or overt, that cause or are the consequence of hypertension. The first suggestion that the kidney could cause hypertension was made in 1836, before arterial pressure could even be measured, by Richard Bright. He observed that cardiac hypertrophy was often present in patients who died of renal disease. It was, however, Goldblatt and his colleagues in 1934 who opened the modern era of experimental and clinical research in renal hypertension. Since then, although far from complete, enthusiastic and intensive research efforts have greatly improved our understanding of the nature of renal hypertension.…

Buch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - .an excellent introduction to clinical nephrology and, therefore, a very helpful text for medical students, house officers, and even beginning fellows in clinical nephrology.' Dialysis and Transplantation, 12: 1 (1983).

Condición: New. pp. x + 303.

Hardcover. 8°. Brauner Halblederband der Zeit mit Lederecken, goldgeprägtem Rückentitel auf blauem Schild sowie Dreiseitenrotschnitt. Der Einband ist etwas berieben. 506 Seiten in fortlaufender Bandpaginierung, jeder Jahresband mit separatem Inhaltsverzeichnis. Erstes Titelblatt mit zwei alten Vorbesitzer-Stempeln (Max Fischer, Magedburg / Münz-Kabinat der Stadt Magdeburg), sonst im Innenbereich sauber und gut erhalten. Band 1 enthält die Nummern 1-9, 2: No. 10-18, 3: 19-28, 4: 29-39, 5; 40-50. -- Inhalt: es werden die Neuerscheinungen aus allen Gebieten der Münzkunde angezeigt und teils auch besprochen/rezensiert bzw. kurz inhaltlich vorgestellt. -- Kategorien: Inhaltsverzeichniss der Numismatischen Zeitschriften / Selbstständige Publikationen und Aufsätze in nicht numismatischen Zeitschriften / Recensions-Verzeichniss, Münz- und Bücher-Verzeichnisse. -- hier einige Beispiele: Francois Lenormant - La monnaie dans l'antique (Paris, 1878-1879) / H. Hantelmann: Antiquarische Miscellen (in: ZS der Gesellschaft für Schleswig-Holstein-Lauenburgische Geschichte, IX. Band, 1879) / Wolff: Die Heckemünze des Grafen Gustav zu Sayn-Wittgenstein zu Clettenberg 1672-1691 (in: ZS des Harz-Vereins für Geschichte und Alterthumskunde, Wernigerode 1879) / K. Th. Eheberg: Über das ältere deutsche Münzwesen und die Hausgenossenschaften besonders in volkswirthschaftlicher Beziehung (Leipzig 1880) / Adolf Hess: Die Siebenbürgischen Münzen des fürstlich Montenuovoschen Münzcabinets (Frankfurt a. M., 1880) / Ferd. Keller: Münzfund im Rennweg, Zürich (in: Anzeiger für Schweizerische Alterthumskunde, Juli 1879) / Theodor Stenzel: Die Vermählungsmedaillen des Anhaltischen Fürstenhauses (Dessau, 1879) / Albert Rehle: Die Münzen der Stadt Kaufbeuren (1880) / W. de Tiesenhausen: Notice sur une collection des monnaies orientales de M. le comte S. Stroganoff (St. Petersburg, 1880) / W. Harster: Versuch einer Speierer Münzgeschichte (aus: Mittheilungen des historischen Vereins der Pfalz, Speier 1882). -- Gegründet wurde die Zeitschrift als 'Literatur-Blatt. Beilage zum Numismatisch-Sphragistischen Anzeiger'. Erstmalig erschienen Nr. 1 am 30. August 1875 bis Mitte des Jahres 1879, danach als selbstständiges Blatt wie hier vorliegend mit der Nummer 1 am 30. Januar 1880. Die Reihe erfuhr wegen der Teilnahme Bahrfeldts am 1. Weltkrieg zwischen Nr. 201 vom Februar 1914 bis zur Nr. 202/203 vom März 1919 eine größere Pause. Bahrfeldt starb am 11. April 1936. Ab Nr. 352/353 Mai 1937 war Herausgeber Gaettens, der auch die Blätter für Münzfreunde herausgab. 1939 wurde das Erscheinen eingestellt, dann vielleicht als Beilage zu Blätter für Münzfreunde? -- Bitte Portokosten außerhalb EU erfragen! / Please ask for postage costs outside EU! / S ' il vous plait demander des frais de port en dehors de l ' UE! // Bitte beachten Sie auch unsere Fotos! / Please also note our photos! / Veuillez noter nos photos -- Leuchtender Herbstbeginn! Wie wäre es mit einem guten Buch? Bei uns werden Sie fündig, stöbern Sie in unserem Ladengeschäft oder online. -- Wir kaufen Ihre werthaltigen Bücher! M12820.…

Relié. Condición: Etat moyen. in-12 Description :308+320+399+272 pp. Dos insolés et tachés, avec des restes d'autocollant. Langue : Français Nb de volumes : 4.

Heftroman 15x22. Condición: Gut. Grossband Blau (Gelbe Seite). 74 Seiten a) 5 Romane altersbedingt leicht gebräunte lichtrandige Exemplare im guten Zustand, 5 Romane 201027745 Sprache: Deutsch Gewicht in Gramm: 400.

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Condición: new. Questo è un articolo print on demand.

Condición: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Blood pressure control is central to all bodily functions. There are many points in the multifaceted cybernetic system wherein hypertension may be produced. Hypertension is a young disorder whose existence has been known for less than a century. It is not.…

Taschenbuch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Blood pressure control is central to all bodily functions. There are many points in the multifaceted cybernetic system wherein hypertension may be produced. Hypertension is a 'young' disorder whose existence has been known for less than a century. It is not only extremely prevalent among every popula tion, but also deleterious to the health of mankind. The more we understand about hypertension's harmful effects, the more urgent is the need for its effective control. The kidney is the central organ that controls vascular tone and body fluid volume; these two factors are dominant in determining arterial blood pres sure. Hence, it is not surprising to find in hypertensive disorders that there are abnormalities in the kidneys, functional or anatomical, subtle or overt, that cause or are the consequence of hypertension. The first suggestion that the kidney could cause hypertension was made in 1836, before arterial pressure could even be measured, by Richard Bright. He observed that cardiac hypertrophy was often present in patients who died of renal disease. It was, however, Goldblatt and his colleagues in 1934 who opened the modern era of experimental and clinical research in renal hypertension. Since then, although far from complete, enthusiastic and intensive research efforts have greatly improved our understanding of the nature of renal hypertension. 324 pp. Englisch.…

Condición: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. 1. Evaluation of patients with renal disease.- 1. Introduction.- 2. Identifying Renal Disease.- 2.1. History.- 2.2. Family history.- 2.3. Physical examination.- 2.4. Urinalysis.- 2.5. The chemical data base.- 2.6. Radiologic techniques.- 2.7. Percutaneous r.…

Taschenbuch. Condición: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Blood pressure control is central to all bodily functions. There are many points in the multifaceted cybernetic system wherein hypertension may be produced. Hypertension is a 'young' disorder whose existence has been known for less than a century. It is not only extremely prevalent among every popula tion, but also deleterious to the health of mankind. The more we understand about hypertension's harmful effects, the more urgent is the need for its effective control. The kidney is the central organ that controls vascular tone and body fluid volume; these two factors are dominant in determining arterial blood pres sure. Hence, it is not surprising to find in hypertensive disorders that there are abnormalities in the kidneys, functional or anatomical, subtle or overt, that cause or are the consequence of hypertension. The first suggestion that the kidney could cause hypertension was made in 1836, before arterial pressure could even be measured, by Richard Bright. He observed that cardiac hypertrophy was often present in patients who died of renal disease. It was, however, Goldblatt and his colleagues in 1934 who opened the modern era of experimental and clinical research in renal hypertension. Since then, although far from complete, enthusiastic and intensive research efforts have greatly improved our understanding of the nature of renal hypertension.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 324 pp. Englisch.…