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Alberico borghetti (11 resultados)

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  • Idioma: Inglés

    Editorial: Springer, 2012

    3662009854 / 9783662009857

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    Librería: GreatBookPrices, Columbia, MD, Estados Unidos de AmericaGreatBookPrices

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    Condición: Usado - Como Nuevo

    EUR 63,95

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    Cantidad disponible: 15 disponibles

    Condición: As New. Unread book in perfect condition.

  • Idioma: Inglés

    Editorial: Springer, 2012

    3662009854 / 9783662009857

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    Librería: GreatBookPrices, Columbia, MD, Estados Unidos de AmericaGreatBookPrices

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    Condición: Nuevo

    EUR 64,55

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    Cantidad disponible: 15 disponibles

    Condición: New.

  • Idioma: Inglés

    Editorial: Springer Verlag, 2012

    3662009854 / 9783662009857

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    Librería: Revaluation Books, Exeter, Reino UnidoRevaluation Books

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    Condición: Nuevo

    EUR 80,95

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    Cantidad disponible: 2 disponibles

    Paperback. Condición: Brand New. reprint edition. 284 pages. 9.20x6.10x0.67 inches. In Stock.

  • Idioma: Inglés

    Editorial: Springer, 2012

    3662009854 / 9783662009857

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    Librería: AHA-BUCH GmbH, Einbeck, AlemaniaAHA-BUCH GmbH

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    Condición: Nuevo

    EUR 66,40

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    Cantidad disponible: 1 disponible

    Taschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - In the last two decades, investigations at the cellular level have progressively gained ground in the context of hypertension research. This choice of approach is due to some extent to the build up of know-how that molecular and cellular biology have been producing at a continuous rate. As the contents list of this volume shows, a large mass of work has been directed to gaining some insight into pathogenetic mechanisms. The pathogenesis of primary hypertension has been progressively categorized as a distinct biological problem, not amenable to the theoretical models that proved successful in understanding the nature of secondary forms of hypertension. At the same time, great efforts have been made to simplify this problem by sorting out, if possible, a few crucial mechanisms from the network of contributory factors in the regulation of blood pressure. The idea that what is to be sought is a primary structural and/or functional fault in arterial muscle has met with widespread acceptance. The strength of this argument lies in the fact that peripheral vascular resistance is increased in all forms of hypertension and, in turn, the diameter of resistance vessels is the dominant factor in the computation of total per ipheral resistance. On the basis of this, cardiovascular structural adaptation was proposed as a positive feedback mechanism tending to maintain hypertension, once begun, whatever the initiating factor is.…

  • Idioma: Inglés

    Editorial: Springer, 2012

    3662009854 / 9783662009857

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    Librería: preigu, Osnabrück, Alemaniapreigu

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    Condición: Nuevo

    EUR 50,65

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    Cantidad disponible: 5 disponibles

    Taschenbuch. Condición: Neu. Cellular Aspects of Hypertension | Alberico Borghetti (u. a.) | Taschenbuch | xii | Englisch | 2012 | Springer | EAN 9783662009857 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.…

  • Idioma: Alemán

    Editorial: Elsevier Science Publ., 1991

    3540539875 / 9783540539872

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    Librería: Martin Preuß / Akademische Buchhandlung Woetzel, Kerzenheim, AlemaniaMartin Preuß / Akademische Buchhandlung Woetzel

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    Miembro de asociación: BOEV

    Condición: Usado - Como Nuevo

    EUR 68,00

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    Cantidad disponible: 1 disponible

    32. Condición: Wie neu. Sprache: Deutsch.

  • Idioma: Inglés

    Editorial: Springer, 2012

    3662009854 / 9783662009857

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    Librería: Brook Bookstore On Demand, Napoli, NA, ItaliaBrook Bookstore On Demand

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    Condición: Nuevo

    EUR 46,22

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    Condición: new. Questo è un articolo print on demand.

  • Idioma: Inglés

    Editorial: Springer 2012-08, 2012

    3662009854 / 9783662009857

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    Librería: Chiron Media, Wallingford, Reino UnidoChiron Media

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    Condición: Nuevo

    EUR 58,95

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    Cantidad disponible: 20 disponibles

    PF. Condición: New. This item is printed on demand. Brand new book, sourced directly from publisher. Dispatch time is 6-7 days from our warehouse. Book will be sent in robust, secure packaging to ensure it reaches you securely.

  • Idioma: Inglés

    Editorial: Springer Berlin Heidelberg Aug 2012, 2012

    3662009854 / 9783662009857

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    Librería: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, AlemaniaBuchWeltWeit Ludwig Meier e.K.

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    Condición: Nuevo

    EUR 53,49

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    Cantidad disponible: 2 disponibles

    Taschenbuch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -In the last two decades, investigations at the cellular level have progressively gained ground in the context of hypertension research. This choice of approach is due to some extent to the build up of know-how that molecular and cellular biology have been producing at a continuous rate. As the contents list of this volume shows, a large mass of work has been directed to gaining some insight into pathogenetic mechanisms. The pathogenesis of primary hypertension has been progressively categorized as a distinct biological problem, not amenable to the theoretical models that proved successful in understanding the nature of secondary forms of hypertension. At the same time, great efforts have been made to simplify this problem by sorting out, if possible, a few crucial mechanisms from the network of contributory factors in the regulation of blood pressure. The idea that what is to be sought is a primary structural and/or functional fault in arterial muscle has met with widespread acceptance. The strength of this argument lies in the fact that peripheral vascular resistance is increased in all forms of hypertension and, in turn, the diameter of resistance vessels is the dominant factor in the computation of total per ipheral resistance. On the basis of this, cardiovascular structural adaptation was proposed as a positive feedback mechanism tending to maintain hypertension, once begun, whatever the initiating factor is. 284 pp. Englisch.…

  • Idioma: Inglés

    Editorial: Springer Berlin Heidelberg, 2012

    3662009854 / 9783662009857

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    Librería: moluna, Greven, Alemaniamoluna

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    Condición: Nuevo

    EUR 48,37

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    Cantidad disponible: Más de 20 disponibles

    Condición: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. In the last two decades, investigations at the cellular level have progressively gained ground in the context of hypertension research. This choice of approach is due to some extent to the build up of know-how that molecular and cellular biology have been p.…

  • Idioma: Inglés

    Editorial: Springer Berlin Heidelberg, Springer Aug 2012, 2012

    3662009854 / 9783662009857

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    Librería: buchversandmimpf2000, Emtmannsberg, BAYE, Alemaniabuchversandmimpf2000

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    Condición: Nuevo

    EUR 53,49

    Envío por EUR 60,00 
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    Cantidad disponible: 1 disponible

    Taschenbuch. Condición: Neu. This item is printed on demand - Print on Demand Titel. Neuware -In the last two decades, investigations at the cellular level have progressively gained ground in the context of hypertension research. This choice of approach is due to some extent to the build up of know-how that molecular and cellular biology have been producing at a continuous rate. As the contents list of this volume shows, a large mass of work has been directed to gaining some insight into pathogenetic mechanisms. The pathogenesis of primary hypertension has been progressively categorized as a distinct biological problem, not amenable to the theoretical models that proved successful in understanding the nature of secondary forms of hypertension. At the same time, great efforts have been made to simplify this problem by sorting out, if possible, a few crucial mechanisms from the network of contributory factors in the regulation of blood pressure. The idea that what is to be sought is a primary structural and/or functional fault in arterial muscle has met with widespread acceptance. The strength of this argument lies in the fact that peripheral vascular resistance is increased in all forms of hypertension and, in turn, the diameter of resistance vessels is the dominant factor in the computation of total per ipheral resistance. On the basis of this, cardiovascular structural adaptation was proposed as a positive feedback mechanism tending to maintain hypertension, once begun, whatever the initiating factor is.Springer-Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 284 pp. Englisch.…