Isbn: 9783540085829 - atherosclerosis - is it reversible? (9 resultados)

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  • Idioma: Inglés

    Editorial: Berlin: Springer, 1978

    3540085823 / 9783540085829

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    Librería: Plurabelle Books Ltd, Cambridge, Reino UnidoPlurabelle Books Ltd

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    Condición: Usado - Bueno

    EUR 64,88

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    Paperback. Condición: Very Good. x 104p paperback, red lettering to front and spine, name to title page, all other pages clean, a fresh copy, well preserved Language: English.

  • Idioma: Inglés

    Editorial: Berlin, Heidelberg, New York : Springer, 1978

    3540085823 / 9783540085829

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    Librería: TschaunersWelt, Waldkirch, AlemaniaTschaunersWelt

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    EUR 67,67

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    (Berlin. Condición: Gut. X, 104 S. : 43 Ill. u. graph. Darst. ; 25 cm Gebraucht, guter bis sehr guter Zustand 2e ISBN: 9783540085829 Sprache: Englisch Gewicht in Gramm: 202.

  • Idioma: Inglés

    Editorial: Springer, 1978

    3540085823 / 9783540085829

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    Librería: Ria Christie Collections, Uxbridge, Reino UnidoRia Christie Collections

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    Condición: Nuevo

    EUR 114,64

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    Cantidad disponible: Más de 20 disponibles

    Condición: New. In English.

  • Idioma: Inglés

    Editorial: Springer Berlin Heidelberg, 1978

    3540085823 / 9783540085829

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    Librería: moluna, Greven, Alemaniamoluna

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    Condición: Nuevo

    EUR 92,27

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    Condición: New.

  • Idioma: Inglés

    Editorial: Springer, 1978

    3540085823 / 9783540085829

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    Librería: Revaluation Books, Exeter, Reino UnidoRevaluation Books

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    Condición: Nuevo

    EUR 151,99

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    Cantidad disponible: 2 disponibles

    Paperback. Condición: Brand New. 1st edition. 114 pages. 9.61x6.69x0.28 inches. In Stock.

  • Idioma: Inglés

    Editorial: Springer Berlin Heidelberg, 1978

    3540085823 / 9783540085829

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    Librería: AHA-BUCH GmbH, Einbeck, AlemaniaAHA-BUCH GmbH

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    Condición: Nuevo

    EUR 106,99

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    Taschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - Even though numerous questions with regard to the pathogenesis of athero sclerosis have not yet been answered, the accumulated evidence indicates significant regression of lesions in experimental animals. This is discussed extensively in this monograph, as are the mechanisms involved in regression of lesions. Whether human atherosclerosis has the potential for regression appears to be the most important, but at the same time the most difficult question to answer. Contrary to experimental atherosclerosis in animals, which can be produced and which can regress within a few months, human lesions in general develop slowly over many years. Therefore, measures aimed at modifying this process may also require many years to be successful. In addition, repeated direct examination of lesions in the human is usually not possible. Nevertheless, recent reports in patients with hyperlipoproteinemias indicate that pronounced and maintained control of hyperlipidemias may lead, even within months, to regression as evidenced by angiography or sophisticated measurements of peripheral circulation. The monograph is divided into two sections. The first will deal with of lipid deposition in the arterial wall, whether 'atherogenesis': mechanisms or not there is evidence of monoclonal origin of human atherosclerosis plaques, cell culture and factors that stimulate smooth muscle proliferation, and animal models of atherogenesis. This section is concluded with a discussion of dietary factors other than lipids in atherogenesis.

  • Idioma: Inglés

    Editorial: Springer, Springer Feb 1978, 1978

    3540085823 / 9783540085829

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    Librería: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, AlemaniaBuchWeltWeit Ludwig Meier e.K.

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    EUR 106,99

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    Taschenbuch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Even though numerous questions with regard to the pathogenesis of athero sclerosis have not yet been answered, the accumulated evidence indicates significant regression of lesions in experimental animals. This is discussed extensively in this monograph, as are the mechanisms involved in regression of lesions. Whether human atherosclerosis has the potential for regression appears to be the most important, but at the same time the most difficult question to answer. Contrary to experimental atherosclerosis in animals, which can be produced and which can regress within a few months, human lesions in general develop slowly over many years. Therefore, measures aimed at modifying this process may also require many years to be successful. In addition, repeated direct examination of lesions in the human is usually not possible. Nevertheless, recent reports in patients with hyperlipoproteinemias indicate that pronounced and maintained control of hyperlipidemias may lead, even within months, to regression as evidenced by angiography or sophisticated measurements of peripheral circulation. The monograph is divided into two sections. The first will deal with of lipid deposition in the arterial wall, whether 'atherogenesis': mechanisms or not there is evidence of monoclonal origin of human atherosclerosis plaques, cell culture and factors that stimulate smooth muscle proliferation, and animal models of atherogenesis. This section is concluded with a discussion of dietary factors other than lipids in atherogenesis. 120 pp. Englisch.

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    Idioma: Inglés

    Editorial: Springer, 1978

    3540085823 / 9783540085829

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    Librería: preigu, Osnabrück, Alemaniapreigu

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    EUR 95,70

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    Taschenbuch. Condición: Neu. Atherosclerosis - is it reversible? | G. Schettler (u. a.) | Taschenbuch | x | Englisch | 1978 | Springer | EAN 9783540085829 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu Print on Demand.

  • Idioma: Inglés

    Editorial: Springer, Springer Feb 1978, 1978

    3540085823 / 9783540085829

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    Librería: buchversandmimpf2000, Emtmannsberg, BAYE, Alemaniabuchversandmimpf2000

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    EUR 106,99

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    Taschenbuch. Condición: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Even though numerous questions with regard to the pathogenesis of athero sclerosis have not yet been answered, the accumulated evidence indicates significant regression of lesions in experimental animals. This is discussed extensively in this monograph, as are the mechanisms involved in regression of lesions. Whether human atherosclerosis has the potential for regression appears to be the most important, but at the same time the most difficult question to answer. Contrary to experimental atherosclerosis in animals, which can be produced and which can regress within a few months, human lesions in general develop slowly over many years. Therefore, measures aimed at modifying this process may also require many years to be successful. In addition, repeated direct examination of lesions in the human is usually not possible. Nevertheless, recent reports in patients with hyperlipoproteinemias indicate that pronounced and maintained control of hyperlipidemias may lead, even within months, to regression as evidenced by angiography or sophisticated measurements of peripheral circulation. The monograph is divided into two sections. The first will deal with of lipid deposition in the arterial wall, whether 'atherogenesis': mechanisms or not there is evidence of monoclonal origin of human atherosclerosis plaques, cell culture and factors that stimulate smooth muscle proliferation, and animal models of atherogenesis. This section is concluded with a discussion of dietary factors other than lipids in atherogenesis.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 120 pp. Englisch.