Idioma: Inglés
Publicado por Kluwer Law International, UK, 2003
ISBN 10: 1402073984 ISBN 13: 9781402073984
Librería: RIVERLEE BOOKS, Waltham Cross, HERTS, Reino Unido
EUR 84,53
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Añadir al carritoHardcover. Condición: Very Good. Very good condition hard cover, some wear to cover and spine, internally clean and tidy with unmarked pages, in a good readable condition.
Librería: GreatBookPrices, Columbia, MD, Estados Unidos de America
EUR 176,43
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Librería: Ria Christie Collections, Uxbridge, Reino Unido
EUR 164,11
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Librería: Ria Christie Collections, Uxbridge, Reino Unido
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EUR 186,43
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Añadir al carritoCondición: As New. Unread book in perfect condition.
Idioma: Inglés
Publicado por Springer International Publishing, 2015
ISBN 10: 3319174363 ISBN 13: 9783319174365
Librería: moluna, Greven, Alemania
EUR 136,16
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Idioma: Inglés
Publicado por Springer International Publishing, 2016
ISBN 10: 331937995X ISBN 13: 9783319379951
Librería: moluna, Greven, Alemania
EUR 136,16
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EUR 191,58
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Añadir al carritoCondición: New.
Librería: Ria Christie Collections, Uxbridge, Reino Unido
EUR 200,69
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EUR 140,00
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Añadir al carritoTaschenbuch. Condición: Neu. Vascular Biochemistry | Peter Zahradka (u. a.) | Taschenbuch | v | Englisch | 2012 | Springer US | EAN 9781461350101 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
Librería: Books Puddle, New York, NY, Estados Unidos de America
EUR 215,09
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Añadir al carritoCondición: New. pp. 746.
Librería: preigu, Osnabrück, Alemania
EUR 140,00
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Añadir al carritoTaschenbuch. Condición: Neu. Cardiac Fibrosis and Heart Failure: Cause or Effect? | Ian M. C. Dixon (u. a.) | Taschenbuch | xv | Englisch | 2016 | Springer | EAN 9783319379951 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
EUR 141,20
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Añadir al carritoTaschenbuch. Condición: Neu. Vascular Biochemistry | Peter Zahradka (u. a.) | Taschenbuch | v | Englisch | 2003 | Springer US | EAN 9781402073984 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
Idioma: Inglés
Publicado por Springer International Publishing, Springer International Publishing, 2016
ISBN 10: 331937995X ISBN 13: 9783319379951
Librería: AHA-BUCH GmbH, Einbeck, Alemania
EUR 160,49
Cantidad disponible: 1 disponibles
Añadir al carritoTaschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - The unique biology of cardiac fibroblasts and related cells, such as cardiac myofibroblasts and valvular interstitial cells, distinguish them from other fibroblastic cells, a concept that is only beginning to be widely appreciated. Further, the natural signals that stimulate and inhibit cardiac fibrosis within these cells are not well understood. This volume compiles articles that address the molecular mechanisms that control the synthesis and secretion of the cardiac ECM. The book showcases chapters that highlight discussion of role of Transforming Growth Factor beta (TGFbeta), an important fibrogenic cytokine and its downstream effectors SMAD in many cardiac diseases. Further, the contributions highlight information to discuss endogenous inhibitors of cardiac fibrosis, as well as advances in tissue engineering specific to matrix in the heart. Finally, discussions of unifying mechanisms of matrix remodeling in valves and myocardium are presented.The mechanisms involved in the stimulation of cardiac fibrosis are not fully understood. In most cases the marginal attenuation of cardiac fibrosis as a result of a given therapy is a beneficial side-effect linked to other primary effects on other cells, especially cardiomyocytes. Very few drugs or agents are known to affect the function and dysfunction of cardiac fibroblasts and myofibroblasts alone. The book helps to translate the information gathered within to allow us to alter the course of fibrogenic events that are typical of cardiac fibrosis, and thereby reduce their burden on the patient and on society itself.
Librería: AHA-BUCH GmbH, Einbeck, Alemania
EUR 160,49
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Añadir al carritoBuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - The unique biology of cardiac fibroblasts and related cells, such as cardiac myofibroblasts and valvular interstitial cells, distinguish them from other fibroblastic cells, a concept that is only beginning to be widely appreciated. Further, the natural signals that stimulate and inhibit cardiac fibrosis within these cells are not well understood. This volume compiles articles that address the molecular mechanisms that control the synthesis and secretion of the cardiac ECM. The book showcases chapters that highlight discussion of role of Transforming Growth Factor beta (TGFbeta), an important fibrogenic cytokine and its downstream effectors SMAD in many cardiac diseases. Further, the contributions highlight information to discuss endogenous inhibitors of cardiac fibrosis, as well as advances in tissue engineering specific to matrix in the heart. Finally, discussions of unifying mechanisms of matrix remodeling in valves and myocardium are presented.The mechanisms involved in the stimulation of cardiac fibrosis are not fully understood. In most cases the marginal attenuation of cardiac fibrosis as a result of a given therapy is a beneficial side-effect linked to other primary effects on other cells, especially cardiomyocytes. Very few drugs or agents are known to affect the function and dysfunction of cardiac fibroblasts and myofibroblasts alone. The book helps to translate the information gathered within to allow us to alter the course of fibrogenic events that are typical of cardiac fibrosis, and thereby reduce their burden on the patient and on society itself.
Idioma: Inglés
Publicado por Springer US, Springer New York, 2012
ISBN 10: 1461350107 ISBN 13: 9781461350101
Librería: AHA-BUCH GmbH, Einbeck, Alemania
EUR 168,73
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Añadir al carritoTaschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - This volume explores all aspects of vascular biochemistry and includes chapters that provide an understanding of vascular function with descriptions of tissue components present in the vascular wall as well as an exploration of the hemodynamic and metabolic activities associated with this function. In addition, some chapters explore the vasculature under conditions which mimic various disease states. The information provided in this volume will provide new insights into the mechanisms that control vascular function as well as therapies designed to treat vascular disease.
EUR 168,73
Cantidad disponible: 1 disponibles
Añadir al carritoTaschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - This volume explores all aspects of vascular biochemistry and includes chapters that provide an understanding of vascular function with descriptions of tissue components present in the vascular wall as well as an exploration of the hemodynamic and metabolic activities associated with this function. In addition, some chapters explore the vasculature under conditions which mimic various disease states. The information provided in this volume will provide new insights into the mechanisms that control vascular function as well as therapies designed to treat vascular disease.
EUR 239,20
Cantidad disponible: 4 disponibles
Añadir al carritoCondición: New. pp. v + 202.
EUR 233,25
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Añadir al carritoPaperback. Condición: Brand New. reprint edition. 207 pages. 11.69x8.27x0.45 inches. In Stock.
Librería: Revaluation Books, Exeter, Reino Unido
EUR 237,84
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Añadir al carritoHardcover. Condición: Brand New. 2015 edition. 454 pages. 9.25x6.10x1.10 inches. In Stock.
Librería: Mispah books, Redhill, SURRE, Reino Unido
EUR 228,58
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Añadir al carritoHardcover. Condición: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.
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EUR 253,58
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Añadir al carritoPaperback. Condición: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.
Librería: GreatBookPrices, Columbia, MD, Estados Unidos de America
EUR 288,58
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Añadir al carritoCondición: As New. Unread book in perfect condition.
Librería: Books Puddle, New York, NY, Estados Unidos de America
EUR 297,71
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Añadir al carritoCondición: New. pp. 452.
Librería: Mispah books, Redhill, SURRE, Reino Unido
EUR 313,11
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Añadir al carritoHardcover. Condición: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.
Librería: Brook Bookstore On Demand, Napoli, NA, Italia
EUR 126,26
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Añadir al carritoCondición: new. Questo è un articolo print on demand.
Librería: Brook Bookstore On Demand, Napoli, NA, Italia
EUR 126,26
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Añadir al carritoCondición: new. Questo è un articolo print on demand.
Librería: Brook Bookstore On Demand, Napoli, NA, Italia
EUR 126,26
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Añadir al carritoCondición: new. Questo è un articolo print on demand.
Idioma: Inglés
Publicado por Springer International Publishing Okt 2016, 2016
ISBN 10: 331937995X ISBN 13: 9783319379951
Librería: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Alemania
EUR 160,49
Cantidad disponible: 2 disponibles
Añadir al carritoTaschenbuch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -The unique biology of cardiac fibroblasts and related cells, such as cardiac myofibroblasts and valvular interstitial cells, distinguish them from other fibroblastic cells, a concept that is only beginning to be widely appreciated. Further, the natural signals that stimulate and inhibit cardiac fibrosis within these cells are not well understood. This volume compiles articles that address the molecular mechanisms that control the synthesis and secretion of the cardiac ECM. The book showcases chapters that highlight discussion of role of Transforming Growth Factor beta (TGFbeta), an important fibrogenic cytokine and its downstream effectors SMAD in many cardiac diseases. Further, the contributions highlight information to discuss endogenous inhibitors of cardiac fibrosis, as well as advances in tissue engineering specific to matrix in the heart. Finally, discussions of unifying mechanisms of matrix remodeling in valves and myocardium are presented.The mechanisms involved in the stimulation of cardiac fibrosis are not fully understood. In most cases the marginal attenuation of cardiac fibrosis as a result of a given therapy is a beneficial side-effect linked to other primary effects on other cells, especially cardiomyocytes. Very few drugs or agents are known to affect the function and dysfunction of cardiac fibroblasts and myofibroblasts alone. The book helps to translate the information gathered within to allow us to alter the course of fibrogenic events that are typical of cardiac fibrosis, and thereby reduce their burden on the patient and on society itself. 452 pp. Englisch.
Idioma: Inglés
Publicado por Springer International Publishing Jul 2015, 2015
ISBN 10: 3319174363 ISBN 13: 9783319174365
Librería: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Alemania
EUR 160,49
Cantidad disponible: 2 disponibles
Añadir al carritoBuch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -The unique biology of cardiac fibroblasts and related cells, such as cardiac myofibroblasts and valvular interstitial cells, distinguish them from other fibroblastic cells, a concept that is only beginning to be widely appreciated. Further, the natural signals that stimulate and inhibit cardiac fibrosis within these cells are not well understood. This volume compiles articles that address the molecular mechanisms that control the synthesis and secretion of the cardiac ECM. The book showcases chapters that highlight discussion of role of Transforming Growth Factor beta (TGFbeta), an important fibrogenic cytokine and its downstream effectors SMAD in many cardiac diseases. Further, the contributions highlight information to discuss endogenous inhibitors of cardiac fibrosis, as well as advances in tissue engineering specific to matrix in the heart. Finally, discussions of unifying mechanisms of matrix remodeling in valves and myocardium are presented.The mechanisms involved in the stimulation of cardiac fibrosis are not fully understood. In most cases the marginal attenuation of cardiac fibrosis as a result of a given therapy is a beneficial side-effect linked to other primary effects on other cells, especially cardiomyocytes. Very few drugs or agents are known to affect the function and dysfunction of cardiac fibroblasts and myofibroblasts alone. The book helps to translate the information gathered within to allow us to alter the course of fibrogenic events that are typical of cardiac fibrosis, and thereby reduce their burden on the patient and on society itself. 452 pp. Englisch.