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Shock Pathogenesis and Therapy: An International Symposium. Stockholm, June 27th–30th 1961 Sponsored by CIBA - Tapa blanda

 
9783662224052: Shock Pathogenesis and Therapy: An International Symposium. Stockholm, June 27th–30th 1961 Sponsored by CIBA
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ISBN 10: 3662224054 ISBN 13: 9783662224052
Editorial: Springer, 2013

Sinopsis

1. Renal failure following circulatory shock develops because of per sistent vasoconstriction which is just sufficient to prevent glomerular filtration. Hypoxia of renal tissue has not been demonstrated in surviving cases. 2. During the low-pressure phase in circulatory shock the remaining blood flow through the medullary regions washes out the osmotic gradient built up by the countercurrent system of Henle’s loops, so that a concentrated urine cannot be formed. 3. During recovery from circulatory failure the osmotic gradient of the medullary region can only be built up if sufficient fluid from the glomerular filtrate reaches the countercurrent system. The greater the GF, the faster the gradient is built up. References 1. BoYLAN, J. W. , and E. AssHAUER: Unpublished data. -2. DEETJEN, P. , and K. KRAMER: Pfliigers Arch. Physiol. (G. ) (in press). - 3. KRAMER, K. , and K. ULLRICH: Pfliigers Arch. Physiol. (G. ) 267,251 (1958). -4. KRA MER, K. , K. THURAU and P. DEETJEN: Pfliigers Arch. Physiol. (G. ) 270, 251 (1960). - 5. KRAMER, K. , and P. DEETJEN: Pfliigers Arch. Physiol. (G. ) 271, 782 (1960). -6. KRAMER, K. , and P. DEETJEN: Unpublished data. - 7. KuHN, W. , and A. RAMEL: Helv. chim. acta 42,628 (1959). - 8. LASSEN, N. A. , 0. MuNcK and J. H. THAYSEN: Acta physiol. Scand. IJ1, 371 (1961). - 9. MUNCK, 0. : Renal Circulation in Acute Renal Failure. Oxford 1958.

"Sinopsis" puede pertenecer a otra edición de este libro.

Reseña del editor

1. Renal failure following circulatory shock develops because of per­ sistent vasoconstriction which is just sufficient to prevent glomerular filtration. Hypoxia of renal tissue has not been demonstrated in surviving cases. 2. During the low-pressure phase in circulatory shock the remaining blood flow through the medullary regions washes out the osmotic gradient built up by the countercurrent system of Henle's loops, so that a concentrated urine cannot be formed. 3. During recovery from circulatory failure the osmotic gradient of the medullary region can only be built up if sufficient fluid from the glomerular filtrate reaches the countercurrent system. The greater the GF, the faster the gradient is built up. References 1. BoYLAN, J. W. , and E. AssHAUER: Unpublished data. -2. DEETJEN, P. , and K. KRAMER: Pfliigers Arch. Physiol. (G. ) (in press). - 3. KRAMER, K. , and K. ULLRICH: Pfliigers Arch. Physiol. (G. ) 267,251 (1958). -4. KRA­ MER, K. , K. THURAU and P. DEETJEN: Pfliigers Arch. Physiol. (G. ) 270, 251 (1960). - 5. KRAMER, K. , and P. DEETJEN: Pfliigers Arch. Physiol. (G. ) 271, 782 (1960). -6. KRAMER, K. , and P. DEETJEN: Unpublished data. - 7. KuHN, W. , and A. RAMEL: Helv. chim. acta 42,628 (1959). - 8. LASSEN, N. A. , 0. MuNcK and J. H. THAYSEN: Acta physiol. Scand. IJ1, 371 (1961). - 9. MUNCK, 0. : Renal Circulation in Acute Renal Failure. Oxford 1958.

"Sobre este título" puede pertenecer a otra edición de este libro.

Editorial
Springer
Año de publicación
2013
Idioma
Inglés
ISBN 10 
3662224054
ISBN 13 
9783662224052
Encuadernación
Tapa blanda
Número de páginas
420
Editor
Bock Klaus D.
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