Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: Basi6 International, Irving, TX, Estados Unidos de America
Condición: Brand New. New. US edition. Expediting shipping for all USA and Europe orders excluding PO Box. Excellent Customer Service.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: Romtrade Corp., STERLING HEIGHTS, MI, Estados Unidos de America
Condición: New. This is a Brand-new US Edition. This Item may be shipped from US or any other country as we have multiple locations worldwide.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: Anis Press, Walnut Creek, CA, Estados Unidos de America
Hardcover Jan 14, 2009. Condición: New.
Publicado por R.G. Landes Co, 1995, 1995
Librería: A Book By Its Cover, Louisville, KY, Estados Unidos de America
Hard Cover. As New. 250 pages.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: Books Puddle, New York, NY, Estados Unidos de America
Condición: New. pp. 292.
Publicado por Austin: R. G. Landes Company, 1995
ISBN 10: 1570592705 ISBN 13: 9781570592706
Librería: Versandantiquariat Waffel-Schröder, Berlin, Alemania
Hardcover/Pappeinband. Condición: Gut. 250 pages. Good. Former library copy with usual markings. Apart from that inside clean. Cover slightly used. Sprache: Englisch Gewicht in Gramm: 770.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: Majestic Books, Hounslow, Reino Unido
Condición: New. pp. 292.
Publicado por Springer, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: GreatBookPrices, Columbia, MD, Estados Unidos de America
Condición: New.
Publicado por Springer, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: booksXpress, Bayonne, NJ, Estados Unidos de America
Soft Cover. Condición: new.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: GreatBookPrices, Columbia, MD, Estados Unidos de America
Condición: New.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: booksXpress, Bayonne, NJ, Estados Unidos de America
Hardcover. Condición: new.
Publicado por Springer, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: Lucky's Textbooks, Dallas, TX, Estados Unidos de America
Condición: New.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: Lucky's Textbooks, Dallas, TX, Estados Unidos de America
Condición: New.
Publicado por Springer, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: Ria Christie Collections, Uxbridge, Reino Unido
Condición: New. PRINT ON DEMAND Book; New; Fast Shipping from the UK. No. book.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: Ria Christie Collections, Uxbridge, Reino Unido
Condición: New. PRINT ON DEMAND Book; New; Fast Shipping from the UK. No. book.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: GreatBookPricesUK, Castle Donington, DERBY, Reino Unido
Condición: New.
Publicado por Springer, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: GreatBookPricesUK, Castle Donington, DERBY, Reino Unido
Condición: New.
Publicado por Springer Netherlands Jan 2009, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Alemania
Buch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably. 296 pp. Englisch.
Publicado por Springer Netherlands Okt 2010, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Alemania
Taschenbuch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably. 292 pp. Englisch.
Publicado por Springer, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: GreatBookPrices, Columbia, MD, Estados Unidos de America
Condición: As New. Unread book in perfect condition.
Publicado por Springer Netherlands, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: moluna, Greven, Alemania
Condición: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Will reflect the recent advances and exponential growth of the field of neurodegenerationWill also provoke questions related to the molecular base and diversity of initiation of conformational diseases up to their pathological classification.
Publicado por Springer Netherlands, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: moluna, Greven, Alemania
Gebunden. Condición: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Will reflect the recent advances and exponential growth of the field of neurodegenerationWill also provoke questions related to the molecular base and diversity of initiation of conformational diseases up to their pathological classification.
Publicado por Springer Netherlands, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: AHA-BUCH GmbH, Einbeck, Alemania
Buch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably.
Publicado por Springer Netherlands, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: AHA-BUCH GmbH, Einbeck, Alemania
Taschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: California Books, Miami, FL, Estados Unidos de America
Condición: New.
Publicado por Springer, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: GreatBookPricesUK, Castle Donington, DERBY, Reino Unido
Condición: As New. Unread book in perfect condition.
Publicado por Springer, 2010
ISBN 10: 9048181275 ISBN 13: 9789048181278
Librería: Revaluation Books, Exeter, Reino Unido
Paperback. Condición: Brand New. 288 pages. 9.25x6.10x0.66 inches. In Stock.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: Revaluation Books, Exeter, Reino Unido
Hardcover. Condición: Brand New. 1st edition. 274 pages. 9.40x6.30x1.00 inches. In Stock.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: GreatBookPricesUK, Castle Donington, DERBY, Reino Unido
Condición: As New. Unread book in perfect condition.
Publicado por Springer, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Librería: Mispah books, Redhill, SURRE, Reino Unido
Hardcover. Condición: Like New. Like New. book.