bacteria complement (33 resultados)

Hardcover. Condición: Good. No Jacket. Pages can have notes/highlighting. Spine may show signs of wear. ~ ThriftBooks: Read More, Spend Less 1.06.

Condición: Good. 191 pp., Hardcover, ex library, else text clean and binding tight. - If you are reading this, this item is actually (physically) in our stock and ready for shipment once ordered. We are not bookjackers. Buyer is responsible for any additional duties, taxes, or fees required by recipient's country.

Condición: Sehr gut. Zustand: Sehr gut | Sprache: Englisch | Produktart: Bücher.

Pas de couverture. Condición: bon. RO40140219: 1964. In-8. En feuillets. Bon état, Livré sans Couverture, Dos satisfaisant, Intérieur frais. Plaquette de 4 pages. Texte sur 2 colonnes. . . . Classification Dewey : 420-Langue anglaise. Anglo-saxon.

Condición: Good. 372 pp., Hardcover, ex library, else text clean and binding tight. - If you are reading this, this item is actually (physically) in our stock and ready for shipment once ordered. We are not bookjackers. Buyer is responsible for any additional duties, taxes, or fees required by recipient's country.

Taschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - Phagocytic cells and complement are probably the most important components of host defense against bacteria which, after overcoming the mucosal and epithelial barriers, multiply in the subepithelial tissue and may threaten to disseminate and invade the blood stream and different organs. Questions concerning the factors which regulate the interactions of the bacterial cell with host defenses are a challenge to research and lead to practical applications for the prevention, treatment and diagnosis of infectious diseases. The questions of expression and regulation of virulence related bacterial genes and gene products, the specific mechanisms of defence reactions by complement and phagocytic cells, their mutual interactions with bacteria and especially bacterial surfaces are focused. Considerations on how to translate this knowledge into the management of infectious diseases are also included.

Hardcover. Condición: Very Good. Estado de la sobrecubierta: No Dust Jacket. 191 pages.

paperback. Condición: New. Ship out in 2 business day, And Fast shipping, Free Tracking number will be provided after the shipment.(Shanghai Science and Technology) complement bacteria and FitnessFour Satisfaction guaranteed,or money back.

Condición: New. pp. 384.

Condición: New. pp. vii + 372.

Condición: New. pp. vii + 372 Illus.

Condición: New. In.

Condición: New. pp. vii + 372.

PF. Condición: New.

Condición: New. In.

Condición: New.

Taschenbuch. Condición: Neu. Druck auf Anfrage Neuware - Printed after ordering - Deficiencies in any of the defense mechanisms of the host can lead to severe microbial infections; these are of clinical relevance. Broad up-to-date knowledge in this field allows identification of many unspecific as well as highly specific defense reactions involved in the struggle against infectious diseases. On the other hand, protective structures on the microbial cell surfaces have been adapted and improve the counterpart's chances of survival. In particular, it has been considered that the great diversity of the bacterial envelopes not only determines the anatomical location of the tissue injury but also induces activation of distinct parts of the complex defense system. The specific defense mechanism, whose most prominent constituent is provided by the antibo.

hardcover. Condición: Good. Connecting readers with great books since 1972! Used textbooks may not include companion materials such as access codes, etc. May have some wear or writing/highlighting. We ship orders daily and Customer Service is our top priority!

Condición: New. pp. 204.

Paperback. Condición: Brand New. reprint edition. 200 pages. 9.60x6.70x0.20 inches. In Stock.

Condición: New.

Hardcover. Condición: Like New. Like New. book.

LeatherBound. Condición: New. Leather Binding on Spine and Corners with Golden leaf printing on spine. Bound in genuine leather with Satin ribbon page markers and Spine with raised gilt bands. A perfect gift for your loved ones. Reprinted from 1919 edition. NO changes have been made to the original text. This is NOT a retyped or an ocr'd reprint. Illustrations, Index, if any, are included in black and white. Each page is checked manually before printing. As this print on demand book is reprinted from a very old book, there could be some missing or flawed pages, but we always try to make the book as complete as possible. Fold-outs, if any, are not part of the book. If the original book was published in multiple volumes then this reprint is of only one volume, not the whole set and contains approximately 26 pages. IF YOU WISH TO ORDER PARTICULAR VOLUME OR ALL THE VOLUMES YOU CAN CONTACT US. Resized as per current standards. Sewing binding for longer life, where the book block is actually sewn (smythe sewn/section sewn) with thread before binding which results in a more durable type of binding. Language: English.

Condición: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Proceedings of the NATO Advanced Research Workshop on Bacteria, Complement and the Phagocytic Cell held in Acquafreda di Maratea, Italy, April 6-9, 1987Phagocytic cells and complement are probably the most important components of host defense against ba.

Condición: New. Print on Demand pp. 384 78 Figures, 67:B&W 6.69 x 9.61 in or 244 x 170 mm (Pinched Crown) Perfect Bound on White w/Gloss Lam.

Taschenbuch. Condición: Neu. This item is printed on demand - Print on Demand Titel. Neuware -InhaltsangabeThe murein sacculus, the bacterial exoskeleton-structure and function in the bacterium and possible role in the host organism.- Chemical nature and cellular location of adhesins of E. coli.- Genetics of O antigen polysaccharide biosynthesis in Shigella and vaccine development.- Capsular polysaccharides (K antigens) of Escherichia coli.- Biogenesis of Gal (1-4)GA1 binding p-pili of uropathogenic E. coli.- The evolution of M proteins of Group A Streptococci.- A new family of pore forming proteins, the perforin family, in immune cytolysis by complement and by lymphocytes.- The molecular basis of the interaction of immunoglobulin G with complement and phagocytic cells.- C3 binding to bacterial surfaces.- The mode of C5B-9 attack on susceptible gram negative bacteria.- Salmonella and complement: the critical influence of 0-polysaccharide within LPS.- Ability to activate the alternative complement pathway as a virulence determinant in Salmonellae.- Interaction of Haemophilus influenzae with complement.- Interaction of Pseudomonas aeruginosa with complement.- Complement-mediated opsonization of group A Streptococci inhibited by the binding of fibrinogen to surface M protein fibrillae.- A surface protein of KLebsiella pneumoniae and Escherichia coli that binds phagocytes and inhibits phagocytosis.- Adhesins, serum resistance and cytolysins of E. coli-genetic structure and role in pathogenicity.- Phagocytosis and intracellular biology of Legionella pneumophila.- Induction of inflammation by Escherichia coli at a mucosal site: requirement for adherence and endotoxin.- IgA-armed T lymphocytes as anti-S. typhi effectors in humans.- Interference of a Staphylococcus aureus bacteriolytic enzyme with polymorphonuclear leucocyte functions.- The opsonic capacity of monoclonal antibodies against Escherichia coli 0111 and its rough mutant E. coli J5.- A mutation in Salmonella typhimurium that enhances resistance to oxygen-independent antimicrobial neutrophil protein.- Plasmid-mediated complement and phagocytosis resistance in E. coli.- The relative role of lipopolysaccharide and capsule in the virulence of E. coli.- Lectinophagocytosis of bacteria mediated by carbohydrate-lectin interactions.- Responses of phagocytosis to bacteria.- Non-secretion of ABO blood group antigens and susceptibility to infectious diseases.Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 384 pp. Englisch.

Taschenbuch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Phagocytic cells and complement are probably the most important components of host defense against bacteria which, after overcoming the mucosal and epithelial barriers, multiply in the subepithelial tissue and may threaten to disseminate and invade the blood stream and different organs. Questions concerning the factors which regulate the interactions of the bacterial cell with host defenses are a challenge to research and lead to practical applications for the prevention, treatment and diagnosis of infectious diseases. The questions of expression and regulation of virulence related bacterial genes and gene products, the specific mechanisms of defence reactions by complement and phagocytic cells, their mutual interactions with bacteria and especially bacterial surfaces are focused. Considerations on how to translate this knowledge into the management of infectious diseases are also included. 384 pp. Englisch.

Condición: New. PRINT ON DEMAND pp. 384.

Condición: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Deficiencies in any of the defense mechanisms of the host can lead to severe microbial infections these are of clinical relevance. Broad up-to-date knowledge in this field allows identification of many unspecific as well as highly specific defense reaction.

Taschenbuch. Condición: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Deficiencies in any of the defense mechanisms of the host can lead to severe microbial infections; these are of clinical relevance. Broad up-to-date knowledge in this field allows identification of many unspecific as well as highly specific defense reactions involved in the struggle against infectious diseases. On the other hand, protective structures on the microbial cell surfaces have been adapted and improve the counterpart's chances of survival. In particular, it has been considered that the great diversity of the bacterial envelopes not only determines the anatomical location of the tissue injury but also induces activation of distinct parts of the complex defense system. The specific defense mechanism, whose most prominent constituent is provided by the antibo 204 pp. Englisch.